This presentation explores the benefits of shared learning between tertiary centres and secondary hospitals to enhance paediatric ultrasound capability. Initiated through collaboration at ASA 2025, the project centred on a one‑week placement at Queensland Children’s Hospital (QCH). This offered valuable exposure to complex pathology, paediatric specialised workflows, and multidisciplinary clinical practice. Key learnings included protocol refinements, practical scanning techniques, and strategies for improving the patient experience in paediatric imaging.
Comparing departments highlighted significant differences in resources, caseload volume, and specialist availability. This underscored the importance of shared learning to bridge capability gaps. Several illustrative cases demonstrate the clinical impact of refined scanning approaches. Additionally, a clinical improvement initiative focused on pyelonephritis assessment revealed further opportunities to enhance diagnostic consistency.
Overall, the presentation emphasises collaboration, the translation of tertiary‑level expertise into smaller hospital settings, and future directions for strengthening paediatric ultrasound services.

Cystic fibrosis is a multi-organ affecting, genetic based disease with ever improving patient outcomes. These patients frequently present for liver surveillance scans, and the purpose of today's talk is to discuss common findings.

In pediatric imaging, ultrasound (USG) is the primary first-line tool for evaluating congenital hepatic lesions due to its safety and real-time assessment of blood flow.

The approach focuses on distinguishing benign developmental masses from rare malignancies using morphological features and vascularity.
Some of the common Benign Hepatic lesions are : Hepatic cysts, Congenital haemangioma, Mesenchymal hamartomas, Biliary hamartomas, focal Nodular hyperplasia. This have to be differentiated from the Malignant Hepatoblastoma.

The presentation will highlight the various characteristics of the lesions on Ultrasound, Colour Doppler and other Radiological investigations.

It will also briefly highlight the follow up and management of these lesions.

Introduction: Our literature review suggests wide variation in accepted pyloric measurements for diagnosis and lack of agreement on which dimension of the pylorus is most accurate for diagnosis of HPS. A correct diagnosis is dependent on sonographer technique and measurement accuracy.
Methods: In a retrospective audit we analysed 161 cases of infants who underwent an ultrasound for suspected hypertrophic pyloric stenosis, with outcomes of evaluating accuracy of sonographic criteria for diagnosis of HPS through correlation with findings at time of surgery. A single observer retrospectively measured the length and transverse diameter of the pyloric canal, and thickness of the pyloric muscle. Measurements were double checked by a second observer and correlated with clinical and surgical findings.
Results: In infants with hypertrophic pyloric stenosis, mean pyloric muscle thickness was 4.7 mm +/- 0.8 mm, pyloric canal length was 19.6 +/-2.4 mm, and pyloric transverse diameter was 13.8 mm +/- 2.0 mm. In patients without hypertrophic pyloric stenosis, pyloric muscle thickness was 1.7 +/- 0.8 mm, pyloric canal length was 10.2 +/- 2.5 mm, and pyloric transverse diameter was 9.5 +/- 2.3 mm.
Conclusions: The mean pyloric measurements of this study are within the range of accepted diagnostic criteria of our institution. Ultrasound diagnosis is 100% congruent with post-surgical diagnosis of HPS. Study showed male predilection and increased likelihood of cases in younger age. No significant separation in measurement for positive and negative cases, and no significant difference in pyloric measurements based on patient age was found.